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2015). These scientific tests, taken jointly, suggest that, although a direct activation of GPR55 or PPAR?? takes place, PEA can create its anti?inflammatory motion within the gut also through oblique activation of CB1 and CB2 receptors, almost certainly as a result of the flexibility of the compound to potentiate the motion of endocannabinoids at

In this review, we discuss the molecular targets of PEA, its pharmacological action in neurological Problems, ache perception and inflammatory health conditions, along with the present evidence within the performance of micronized and extremely?micronized formulations of PEA when presented alone or in combination While using the antioxidant flavono

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